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How Artificial Sweeteners Suppresses Thyroid Function Induce Diabetes and Obesity Related Changes
How artificial sweeteners suppresses thyroid function, induce diabetes and obesityrelated Changes By:Sayer Ji, Founder A groundbreaking new study published in the prestigious journal Nature has revealed how noncaloric artificial sweeteners (NAS) drive obesity and diabetesrelated changes in both mice and humans.
The study titled, artificial sweeteners induce glucose intolerance by altering the gut microbiota, states: Here we demonstrate that consumption of commonly used NAS formulations drives the development of glucose intolerance through induction of compositional and functional alterations to the intestinal microbiota. Part 1: Gut Bacteria Mediate Artificial SweetenerInduced Blood Sugar Disturbances.
The researchers administered commercially available formulations of saccharin (sweet N Low), sucralose (Splenda) or aspartame (Equal) in the drinking water of 10week old mice. The control group received I either glucose or sucrose. After 11 weeks, the three artificial sweetener fed groups developed marked glucose intolerance, with saccharin having the most deleterious effects followed by sucralose and aspartame. Gut changes linked to artificial sweeteners.
Graph showing the effects of artificial sweeteners on blood glucose levels. The researchers hypothesized that since artificial sweeteners pass through the gut mostly unabsorbed by the body that the microbiota may be responsible for regulating their observed adverse effects on blood sugar. To test their theory they administered antibiotics to mice while keeping them on their diet and sweetener supplementation regimens.
Their results indicate that gut bacteria indeed drive the adverse effects of these sweeteners: Notably, after 4 weeks of antibiotic treatment, differences in glucose intolerance between NASdrinking mice and controls were abolished both in the lean and the obese states. Similar effects were observed with the Grampositivetargeting antibiotic vancomycin (antibiotics B, 0.5 g l1). These results suggest that NASinduced glucose intolerance is mediated through alterations.
To the commensal microbiota, with contributions from diverse bacterial taxa. Finally, in order to test whether the role of microbiota in upsetting blood sugar balance was cause and effect they performed a fecal transplant from mice receiving saccharin or glucose into germfree mice receiving the same normalchow diet. Their results confirmed the crucial role of the microbiota in inducing blood sugar disturbing effects:.
notably, recipients of microbiota from mice consuming commercial saccharin exhibited impaired glucose tolerance as compared to control (glucose) microbiota recipients, determined 6 days following transfer. Transferring the microbiota composition of HFDconsuming mice drinking water or pure saccharin replicated the glucose intolerance phenotype. Together, these results establish that the metabolic derangements induced by NAS consumption.
Are mediated by the intestinal microbiota. artificial sweeteners induce negative changes In Gut Bacteria The researchers next performed an analysis of the changes induced in the composition of microbiota by saccharin finding a widespread reorganization: Compared to all control groups, the microbiota of saccharinconsuming mice displayed considerable dysbiosis, with more than 40 operational taxonomic units (OTUs) significantly altered in abundance.